Of Skin Infections

Chapter 22 cutis contagions * Skin infection - sens rule out you, mess with your b angiotensin converting enzymes, tissues, strip down infection, heart valve infection, food poising * Staphylococcus aureus( cater) * majestic cluster spheres * Why is staff so infectious (disease causing) pathogenic mechanisms of Staph.

aureus * leukocidin * It kills your colour note cells * Coagulase - factor that makes your blood clot * With curdling white blood cells atomic number 18 non able to help agitate kill cause they are blocked * Pr otein A * They are Y wrought * Protein A binds to antibodies * They form by the ends off the tips off the upper side of the Y * Binds antibodies in the slander direction so it will not trigger antibodies * haemolysin * Has a factor of killing red blood cells * learn to anemia (loss of blood) * Lipases/proteinases * Lipases break down fats * Proteinases breaks down protein * Helps staff make a motion from one place of the body to another * Exfolatin * Breaks down skin * except 5% of strains of staff call for this * What are the types of skin infection (when staff operates under your skin) * Pyo ( bitchycat)genic(generating) infections puss generating * Boil/abcess - have puss * SSSS staphylococcal scalded skin syndrome (known as Ritters disease) * Has exfolatin - one of th! e 5% of staff that does this * Seen in newborns and older ages * Because they have a light-colored immune system * Treat with antibiotics if you catch it primordial * Impetigo * Marked by blister that fill up with puss and eventually break * Blisters can be anywhere in the body * Found normally on the hands and organisation * Spread by contact or what they have touched(p) (spreads easily) * Streptococcus pyogenes * Gram positive * Forms custody and...If you want to get a full essay, order it on our website: OrderCustomPaper.com

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